Comparison of Two Assays for Molecular Determination of Rifampin Resistance in Clinical Samples from Patients with Buruli Ulcer Disease

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dc.contributor.authorPhillips, Richard Odame
dc.contributor.authorJansson, Moritz
dc.contributor.authorBeissner, Marcus
dc.contributor.authorBadziklou, Kossi
dc.contributor.authorPiten, Ebekalisai
dc.contributor.authoret. al
dc.date.accessioned2020-01-16T15:54:22Z
dc.date.accessioned2023-04-19T01:49:44Z
dc.date.available2020-01-16T15:54:22Z
dc.date.available2023-04-19T01:49:44Z
dc.date.issued2014-01-29
dc.descriptionAn article published by Journal of Clinical Microbiology and available doi.org/10.1128 /JCM.03119-13.en_US
dc.description.abstractBuruli ulcer disease (BUD), caused by Mycobacterium ulcerans, involves the skin, subcutaneous fatty tissue, and bones and predominantly affects children 15 years of age. If left untreated, contractures may cause severe functional limitation. Standardized antimycobacterial treatment consists of rifampin (RMP) and streptomycin administered for 8 weeks. An oral regimen combiningRMPand clarithromycin is currently under clinical evaluation (1–3). Notwithstanding the efficiency of chemotherapy, treatment failures and various types of secondary lesions have been reported, suggesting the need for customized clinical management strategies (4–6).en_US
dc.description.sponsorshipKNUSTen_US
dc.identifier.citationJournal of Clinical Microbiology Volume 52 p. 1246–1249en_US
dc.identifier.urihttps://ir.knust.edu.gh/handle/123456789/11947
dc.language.isoenen_US
dc.publisherJournal of Clinical Microbiologyen_US
dc.titleComparison of Two Assays for Molecular Determination of Rifampin Resistance in Clinical Samples from Patients with Buruli Ulcer Diseaseen_US
dc.typeArticleen_US
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